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Given the critical role played by NOX in AD, there is potential for selectively targeting specific NOX subtypes to rectify eNOS uncoupling and mitochondrial dysfunction, thereby offering a viable approach for the prevention and treatment of AD ( from the body, diminish the infiltration of inflammatory cells, and retard the aging process (185)

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Without adequate collagen production and proper organization of collagen fibers, wounds heal with reduced strength and may be more prone to reopening or developing problematic scars

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