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Candidate genes such as MIKC-type MADS domain proteins and calmodulin-binding transcription factors have been emphasized for their involvement in salt stress signaling (Leawtrakun et al., 2024a)
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In this context, a previous work using Drosophila demonstrated that dZIP7 dysfunction causes the accumulation of Notch and EGFR in cells by impairing the proteasome machinery involved in ERAD 37
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